A.I.M.'s Treatment Focus

Reparation of Cells

Just as our bodies have the ability to kill pathogens invading our bodies and cells that

become cancerous, our bodies possess the inherent ability to repair damage incurred

at the cellular level. When we fast for several days the lack of nutrients trigger a gene

called mTor to down regulate, or shut off. This gene in this state triggers the body to

go into a catabolic mode and triggers autophagy.

mTor, which stands for the mammalian target of rapamycin, is responsible for telling

our cells what to do in correlation with the level of nutrients in the blood, mTor can

dictate whether our cells, grow, remain static, die or repair themselves. When an

adverse situation such as starvation comes about, mTor responds by down regulating ,

thereby triggering mTor to communicate to other cells its time to go into repair mode.

Lack of nutrients is not the only trigger for atophogy, stress can also trigger the same

response.

Autophagyt, without doubt, is an evolutionary trait arising from natural selection and appears to be designed to make the organism stronger as a response, or adaptiation, to environmental stress or periods of famine. This trait manifests itself in many organisms that survive in harsh conditions. It is long evident that organisms that live in sea were adverse conditions not only tend to live longer, they live with a remarkable abilities to stay healthy and ward off cancer and other age related disease.

At AIM we utilize a combination of FDA approved drugs combined with other supplements in order to facilitate autophagy and control certain side effects. This treatment has been shown to restore function in several types of cells translating to improved metrics in several health indices. In addition, it has also been shown to ward off cancer at every stage by preventing or impeding angiogenisis, as well as inducing apoptosis (the process of inducing cellular death of cancer cells.

Regenerating the Thymus

We have a gland in the center of our chest called the thymus. This gland is responsible for teaching juvenile T cells what to protect and what to attack and kill. This gland is part of our adaptive immune system and plays a critical role.

The Thymus starts down a long road of degradation beginning right after puberty when it begins to shrink. Over the next few decades the thymus becomes more and more enveloped in fat cells which serve no functional use. By the time we are in our 60's the thymus has been reduced to a ball of fat and looses its critical function and results in premature T cells and cells that cause auto immune disease by attacking our own cells began proliferating casusing several concerning health problems.

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